Galactopoiesis and Milk Ejection in Veterinary Science
GALACTOPOIESIS
Dr Pramod Kumar
Bihar Veterinary College
Bihar Animal Sciences University
Patna
Progesterone and estrogen stimulate
proliferation of secretory tissues. In vivo,
sequential addition of insulin, glucocorticoid
and prolactin leads to biosynthesis of casein
and lactose. Progesterone inhibits
differentiation of secretory cells. Sensitivity of
individual cells to progestational inhibition
may decrease variably which may be
interdependent upon relative increases in
estrogen, prolactin, corticoids and growth
hormone to cause asynchronies at calving.
Galactopoiesis
(maintainance of lactation)
•
Prolactin
•
GH
•
Cortisol
•
PTH
•
Insulin
•
Oxytocin
Ejection of Milk: Oxytocin
•
Milk let down reflex or suckling reflex
•
Neuroendocrine reflex
Suckling of teats
Afferent conduction of APs
spinal cord
hypothalamus
Prolactin
secretion
Oxytocin
secretion
Increase milk in the alveoli
Contraction of the myoepithelial cells
Ejection of milk
Role of hormones
•
Estrogen & Progesterone
specific effect of both these
hormones is to inhibit the actual secretion of milk
•
Prolactin: stimulates milk production
•
Oxytocin: stimulates milk release
•
Human chorionic somatomammotropin: lactogenic
properties and support action of prolactin
Galactopoiesis is the process of maintaining lactation in animals, involving hormones like prolactin, GH, cortisol, PTH, insulin, and oxytocin. This process also includes the role of progesterone, estrogen, and other hormones in milk production and release. Milk ejection, facilitated by oxytocin, involves a neuroendocrine reflex leading to milk let-down during suckling. Understanding these processes is crucial for proper animal care and management in veterinary science.
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Presentation Transcript
GALACTOPOIESIS Dr Pramod Kumar Bihar Veterinary College Bihar Animal Sciences University Patna
Progesterone proliferation of secretory tissues. In vivo, sequential addition of insulin, glucocorticoid and prolactin leads to biosynthesis of casein and lactose. Progesterone differentiation of secretory cells. Sensitivity of individual cells to progestational inhibition may decrease variably interdependent upon relative increases in estrogen, prolactin, corticoids and growth hormone to cause asynchronies at calving. and estrogen stimulate inhibits which may be
Galactopoiesis (maintainance of lactation) Prolactin GH Cortisol PTH Insulin Oxytocin
Ejection of Milk: Oxytocin Milk let down reflex or suckling reflex Neuroendocrine reflex spinal cord Suckling of teats Afferent conduction of APs Contraction of the myoepithelial cells Oxytocin secretion hypothalamus Ejection of milk Prolactin secretion Increase milk in the alveoli
Role of hormones Estrogen & Progesterone specific effect of both these hormones is to inhibit the actual secretion of milk Prolactin: stimulates milk production Oxytocin: stimulates milk release Human chorionic somatomammotropin: lactogenic properties and support action of prolactin
